β-cell deficient mouse models reveal that glucose and KATP channel blockers control glucagon secretion independently of β-cell derived paracrine factor

Chae, Heeyoung;Lai, Bao Khanh;Gomez Ruiz, Ana;Antoine, Nancy;Gilon, Patrick;et.al.
(2016) 52nd European Association for the Study of Diabetes (EASD) Annual Meeting — Location: (Germany) Munich (12.September.2016)

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  • Chae, HeeyoungUCLouvain
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  • Lai, Bao KhanhUCLouvain
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  • Gomez Ruiz, AnaUCLouvain
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  • Antoine, NancyUCLouvain
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  • Gilon, Patrickorcid-logoUCLouvain
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Chae, H., Lai, B. K., Gomez Ruiz, A., Antoine, N., Herrera, P., & Gilon, P. (2016). β-cell deficient mouse models reveal that glucose and KATP channel blockers control glucagon secretion independently of β-cell derived paracrine factor. Diabetologia : clinical and experimental diabetes and metabolism, 59(Suppl 1), S256. https://hdl.handle.net/2078.5/181059 (Original work published 2016)