Atypical mechanisms of Ca2+-release from the endoplasmic reticulum contribute to the cytosolic Ca2+ rise induced by depolarization in mouse ß-cells

Beauvois, Mélanie;Arredouani, Abdelilah;Rolland, Jean-François;Jonas, Jean-Christophe;Gilon, Patrick;et.al.
(2003) 18th Internatioal Diabetes Federation Congress (IDF) — Location: (France) Paris (24.August.2003)

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Authors
  • Beauvois, MélanieUCLouvain
    Author
  • Arredouani, AbdelilahUCLouvain
    Author
  • Rolland, Jean-FrançoisUCLouvain
    Author
  • Jonas, Jean-Christopheorcid-logoUCLouvain
    Author
  • Henquin, Jean-ClaudeUCLouvain
    Author
  • Gilon, Patrickorcid-logoUCLouvain
    Author
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Abstract
BACKGROUND AND AIMS: In pancreatic ß-cells, Ca2+ influx through voltagedependent Ca2+ channels plays a prominent role in the depolarizationinduced rise in free cytosolic Ca2+ concentration ([Ca2+]c). The contribution of Ca2+ release from the endoplasmic reticulum (ER) to this [Ca2+]c elevation is largely debated and was investigated here [...]
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Beauvois, M., Arredouani, A., Rolland, J.-F., Jonas, J.-C., Henquin, J.-C., & Gilon, P. (2003). Atypical mechanisms of Ca2+-release from the endoplasmic reticulum contribute to the cytosolic Ca2+ rise induced by depolarization in mouse ß-cells. Diabetologia : clinical and experimental diabetes and metabolism, 46(Suppl 2), A160. https://hdl.handle.net/2078.5/119034 (Original work published 2003)