Neurological characterization of mice deficient in GSK3α highlight pleiotropic physiological functions in cognition and pathological activity as Tau kinase.
(2013) Molecular Brain —
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- Abstract
- GSK3α proved to be regulated independently from GSK3β, and to exert non-redundant physiological neurological functions in general behavior and in cognition. Moreover, GSK3α contributes to the pathological phosphorylation of protein Tau.
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Maurin, H., Lechat, B., Dewachter, I., Ris, L., Louis, J. V., Borghgraef, P., Devijver, H., Jaworski, T., & Van Leuven, F. (2013). Neurological characterization of mice deficient in GSK3α highlight pleiotropic physiological functions in cognition and pathological activity as Tau kinase. Molecular Brain. https://doi.org/10.1186/1756-6606-6-27